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Targeting Endothelial Erk1/2-Akt Axis as a Regeneration Strategy to Bypass Fibrosis during Chronic Liver Injury in Mice

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Answer: GHK-Cu appears to work as a signaling peptide, prompting fibroblasts and other cells to ramp up collagen, elastin, and antioxidant activity, while also influencing the expression of many genes involved in tissue maintenance

Redox status is altered in G6PD deficient RBCs, supporting the idea that G6PD deficiency confers protection against malarial infection (although predisposes to hemolysis)