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The researchers exposed both naturally aging mice and mice genetically modified to age more rapidly to toxic chemotherapy drugs to induce additional stress on their cells

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The review underscores the role of irregular activation of the HGF/c-MET signaling route in facilitating communication between CAFs and various immune elements, modifying ECM components, attracting and integrating immunosuppressive cells (MDSCs, TAMs, and Tregs), and controlling PD-L1 protein expression

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