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Consequently, mitophagy serves to protect neurons from damage in HD, and a deficiency in this process contributes to further malignant deterioration of HD pathology

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While it doesn't directly participate in the glutathione antioxidant defense system, it is essential for the synthesis and metabolism of amino acids, including cysteine

GSH-deficient FoB upregulate glycolysis but accumulate defective mitochondria Based on our scRNA analyses and the effect of Gclc deficiency on glucose uptake (Fig