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It also increased recovery of neurons in the damaged myenteric plexus in two in vivo models of enteric neuronal damage in mice, specifically the damage resulting from administration of a high-fat diet, or the serosal application of the cationic detergent, benzalkonium chloride ( The potential for neurogenesis and nerve growth factors to restore normal propulsion has been demonstrated in both animal and human studies

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One of the key mechanisms through which nicotinamide may exert these protective effects is by restoring the levels of nicotinamide adenine dinucleotide (NAD+), which is critical for mitochondrial metabolism and energy production