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Ipamorelin-induced GH release drives hepatic IGF-1 production and IGF-1 axis activation through the same GH-IGF-1 axis biology engaged by non-selective GH secretagogues and but does so without prolactin-mediated IGF-1 production confounds that arise from GHRP-6 and GHRP-2s co-stimulation of pituitary prolactin

One key difference between the two is how they work: Tirzepatide is a dual agonist, which means it affects two different pathways, GLP-1 and GIP, whereas semaglutide only affects GLP-1

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The outcome looks natural, refreshed, and proportional to the patients body rather than looking obvious or overdone