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Hodge, R

Because NAD + reduction is frequently accompanied by impaired mitochondrial performance concomitant to tissue damage and dysfunction [20, 21], tissue NAD + restoration has been proposed as a promising therapeutic target in mitochondria-driven strategies to treat metabolic diseases [19]

Activity of four allelic forms of glutathione S-transferase hGSTP1-1 for diol epoxides of polycyclic aromatic hydrocarbons

However, preclinical studies demonstrate therapeutic effects with once-daily or even less frequent administration, suggesting tissue distribution, receptor binding, or cellular signaling effects extend beyond plasma clearance

The many faces of vitamin B12: catalysis by cobalamin-dependent enzymes