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The above data suggest that the neurodevelopmental rat model of schizophrenia induced by glutathione deficit evoked by repeated treatment with BSO alone and together with GBR 12909 in early postnatal life may be very important for studies on the pathomechanism of schizophrenia

Cancer Cell Internat

[PubMed] [Google Scholar] Henneberg A, Riedl B, Dumke HO, Kornhuber HH

The first involves the development of single-arm humanized antibodies to HGF or Met

De nombreuses tudes ont montr que le glutathion pouvait tre appliqu localement sans inquitude, il n'y a donc aucune raison de craindre des effets secondaires